On PubMed you can find the study:
Research Centre for Endocrinology and Metabolism, Sahlgrenska University Hospital, Göteborg, Sweden.
- Point 1: The association of several risk factors including, obesity, dyslipoproteinemia, hepatic steatosis, insulin resistance and hypertension with Type 2 (non-insulin-dependent) diabetes mellitus and myocardial infarction has long been known and has been termed the “metabolic syndrome”.
- Point 2: Type 2 diabetes and cardiovascular morbidity is the mass of intraabdominal fat. Striking similarities exist between the metabolic syndrome and untreated growth hormone (GH) deficiency in adults.
- Point 3: The main association is abdominal/visceral obesity and insulin resistance. Other features common to both conditions are premature atherosclerosis and increased mortality from cardiovascular diseases. These similarities indicate that undetectable and low levels of GH may be of importance in the metabolic aberrations observed in both these conditions.
- Point 4: Nine months of GH treatment reduced their total body fat and resulted in a specific and a marked decrease in both abdominal subcutaneous and visceral adipose tissue. Moreover, insulin sensitivity improved and serum concentrations of total cholesterol and triglyceride decreased. Diastolic blood pressure also decreased. The finding that GH replacement in men with abdominal obesity can diminish the negative metabolic consequences of visceral obesity suggests that low levels of this hormone are of importance for the metabolic aberrations associated with visceral/abdominal obesity.
Ref: https://www.ncbi.nlm.nih.gov/pubmed/10442570
See our Blog on Reversing Metabolic Syndrome <Click here>.
Also see: https://www.hindawi.com/journals/mi/2010/434562/
It is important to note that there is also conflicting evidence regarding growth hormone and insulin resistance. But in recent years, a better understanding of metabolism, in general, helps us understand the flaws in prior conflicting studies with growth hormone and insulin resistance.
With more knowledge about intermittent fasting, ketogenic dieting, and fasting-mimicking dieting, and their role at successfully managing hyperglycemia, the role of carbohydrates and sugar metabolism is most likely more of a factor. By managing post-prandial glucose following meals using a $30 glucose meter, most can eat a whole food diet and limit carbohydrate intake at a level that prevents excessive glucose.
Dr. Jason Fung and others have elucidated this subject very well. See the following YouTube video for more information:
Jason Fung, MD:
The combination of proper dietary protocols with HGH is the way future research studies should be designed for reversing metabolic syndrome in patients.